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What I found particularly interesting is the reminder that diseases with similar symptoms do not necessarily share the same biology

Current hypotheses regarding the pathophysiology of anxiety disorders emphasize an imbalance between neuronal excitation and inhibition, characterized by reduced Gamma-aminobutyric acid (GABA) inhibition and excessive glutamatergic activity, which contributes to neuronal hyperexcitability and the manifestation of anxious behaviors (111)

Rare GAT1 mutations can cause epilepsy.[ref] GABA beyond the brain: immune system, pancreas, gut Much of what you read about GABA focuses on the brain and how GABA affects neurotransmitters, mood, and behavior

Medication is a powerful tool for the people who need it, and food is a powerful supporting cast for everyone else

Joint discomfort and numbness become real concerns at these doses

Without that capillary supply, GHK-Cu has the signal but no nutrient delivery to the treatment site